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Thus, in testosterone deficiency, lipolysis is inhibited, favoring the accumulation of adipose tissue [6 ], which is reversed by testosterone administration. In addition, it has been reported that in hypogonadal patients, the deposition of visceral adipose tissue leads in turn to a further decrease in testosterone concentrations via conversion to estradiol by the aromatase. This leads to further abdominal fat deposition and a higher testosterone deficiency [4. In parallel, hyperinsulinemia is associated with decreased SHBG production, which decreases plasma total testosterone [67 ]. To date, the question of whether hypogonadism influences insulin resistance by increased abdominal obesity or whether obesity favors the reduction of plasma testosterone concentrations is still debated. However, insulin resistance leads to increased risk factors including increased triglycerides, lower HDL, and predominance of LDL-C. To these lipoprotein factors are added intolerance to carbohydrates, high blood pressure, and pro-coagulant and antifibrinolytic states [68 ].
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